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Estrogen can activate the transcription of NO synthase by binding to the classical E2 receptor or GPER (Iorga et al., 2017), producing the vasodilator, NO, which spreads to vascular smooth muscle cells, activates guanylate cyclase, and increases the levels of cyclic guanosine phosphate ( Deficiencies in estrogen therapy and challenges in clinical treatment Owing to their numerous biological effects, as shown in Figure 3, estrogen and its analogs are associated with the treatment of a wide range of diseases, including neuroprotective therapies for ischemic stroke, Alzheimer’s disease, and Parkinson’s disease

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Lichtenstein P et al